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Fig. 1 | Epigenetics & Chromatin

Fig. 1

From: Multi-omics analyses of MEN1 missense mutations identify disruption of menin–MLL and menin–JunD interactions as critical requirements for molecular pathogenicity

Fig. 1

Selection of MEN1 missense mutation-derived forms of menin. A Nine disease-relevant MEN1 missense mutations were selected based on their predicted stability in silico and ability to bind to MLL1 and/or JunD. The predicted effects of the mutations are listed. B The nine MEN1 mutations are projected on the menin structure

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Epigenetics & Chromatin

ISSN: 1756-8935