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Fig. 1 | Epigenetics & Chromatin

Fig. 1

From: Developmental programming of DNA methylation and gene expression patterns is associated with extreme cardiovascular tolerance to anoxia in the common snapping turtle

Fig. 1

Effects of acute anoxia and reoxygenation on haemodynamic variables from N21 and H10 turtles. Turtles from the N21 (red circles, n = 6) and H10 (blue squares, n = 5) cohorts were subjected to 120 min of anoxia followed by 30 min reoxygenation. A Heart rate (\(f_{{\text{H}}}\)), B systemic blood flow (\(\dot{Q}_{{{\text{Sys}}}}\)), C pulmonary blood flow (\(\dot{Q}_{{{\text{Pul}}}}\)), D total blood flow (\(\dot{Q}_{{{\text{Tot}}}}\)), E systemic stroke volume (\(V_{{{\text{S}},{\text{Sys}}}}\)), F pulmonary stroke volume (\(V_{{{\text{S}},{\text{Pul}}}}\)), G shunt distribution (\(\dot{Q}_{{{\text{Shunt}}}}\)), H shunt ratio (\({{\dot{Q}_{{{\text{Pul}}}} } \mathord{\left/ {\vphantom {{\dot{Q}_{{{\text{Pul}}}} } {\dot{Q}_{{{\text{Sys}}}} }}} \right. \kern-\nulldelimiterspace} {\dot{Q}_{{{\text{Sys}}}} }}\)), I mean ventricular pressure (\(P_{{{\text{Vent}}}}\)), and J cardiac power output. Values are mean ± SEM, asterisks (*) indicate statistically significance difference between N21 and H10 groups, dollar ($) and psi (Ψ) symbols denote a significant difference between that data point and pre-anoxic levels (time zero) in the N21 and H10 groups, respectively (p ≤ 0.05)

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